By Myron Yanoff
Revised and increased, the second one variation of this atlas offers enlargements of the numerous photographs in addition to new sections on immunohistopathology, AIDS, and problems. color illustrations supplement the textual content, which examines uncomplicated rules of pathology, congenital anomalies, nongranulomatous irritation, sufrgical and nonsurgical trauma, lids and lacrimal drainage platforms, conjunctiva, cornea and sclera, uvea, retina, vitreous, optic nerve, diabetis mellitus, glaucoma, ocular melanoic tumors, retinoblastoma, and pseudoglioma.
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Additional info for Ocular pathology : a color atlas
These areas of nonfunctional capillaries are demonstrated angiographically to be non-perfused. If an area of non-perfusion becomes quite large, it may appear as cotton-wool spots. Pre-Proliferative Diabetic Retinopathy Between non-proliferative and proliferative retinopathy, there is a pattern of retinal changes that has been called preproliferative diabetic retinopathy. These changes such as soft exudates (cotton-wool spots), venous abnormalities and intraretinal microvascular abnormalities (IRMA), can be well demonstrated by angiography as a flat network of tortuous capillaries that do not follow the normal capillary network and do leak (Figure 11) Proliferative Diabetic Retinopathy The appearance of new vessel formation either on the disc or elsewhere in the retina signals a proliferative diabetic retinopathy.
Other conditions which show hyperfluorescence are central serous chorioretinopathy, in which the dye accumulates between the neurosensory retina and the RPE to produce a diffuse region of hyperfluorescence. In ocular histoplasmosis, nodular elevation of the RPE by choroidal neovascular membranes may cause choroidal hyperfluorescence. Hypofluorescence The absence of fluorescein in a location where it is normally found may be attributable to either a lack of perfusion or the absence of tissue itself.
A) Fluorescein angiography shows the late hyperfluorescence corresponding to the macular lesion. b) Optical Coherence Tomography (OCT) shows how neovascularization is causing a thickening of retina layers with presence of intraretinal fluid and little neuroepithelial detachment, confirming the activity of the lesion. b In Figure 1a-b we show the normal retina, for comparison with abnormal cases. Figure 2a-b shows alterations in age-related macular degeneration with choroidal neovascularization.
Ocular pathology : a color atlas by Myron Yanoff