By Stanley H. Rosenbaum MD MA, Lewis J. Kaplan
Administration of Peri-operative issues is tested within the factor of Surgical Clinics, visitor edited Drs. Lewis Kaplan and Stanley Rosenbaum. subject matters include: fluids and electrolytes, hypoperfusion, surprise states and ACS, surgical prophylaxis and hassle avoidance bundles, NSQIP, SCIP and TQIP, post-operative malnutrition and probiotic treatment, post-operative malnutrition and probiotic remedy, harm keep an eye on for intra-abdominal sepsis, multi-drug resistant organisms and antibiotic administration, pneumonia and acute pulmonary failure, organ failure avoidance and mitigation techniques, delirium, EtOH withdrawl and polypharmacy withdrawl states, mobile and molecular body structure of nerve damage and chronic post-operative ache, acute ache administration following operation: combating continual post-operative ache, immense transfusion and comparable matters, post-op ileus, SBO and colonic dysmotility, GI hemorrhage, and in-hospital rescue cures.
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Additional resources for Management of Peri-operative Complications, An Issue of Surgical Clinics, 1e
4. Hai SA. Permissive hypotensive resuscitation—an evolving concept in trauma. J Pak Med Assoc 2004;54(8):434–6. 5. Rivers E, Nguyen B, Havstad S, et al. Early goal-directed therapy in the treatment of severe sepsis and septic shock. N Engl J Med 2001;345(19):1368–77. 6. Crookes BA, Cohn SM, Bloch S, et al. Can near-infrared spectroscopy identify the severity of shock in trauma patients? J Trauma 2005;58(4):806–13. 7. Cohn SM, Nathens AB, Moore FA, et al. Tissue oxygen saturation predicts the development of organ dysfunction during traumatic shock resuscitation.
36. Kaplan LJ, Kellum JA. Initial pH, base deficit, lactate, anion gap, strong ion difference, and strong ion gap predict outcome from major vascular injury. Crit Care Med 2004;32(5):1120–4. 37. Kaplan LJ, Kellum JA. Comparison of acid-base models for prediction of hospital mortality after trauma. Shock 2008;29(6):662–6. 38. Kaplan LJ, Philbin N, Arnaud F, et al. Resuscitation from hemorrhagic shock: fluid selection and infusion strategy drives unmeasured ion genesis. J Trauma 2006; 61(1):90–7.
First, as originally described by Coombs,17 intrathoracic pressure (ITP) is increased as a result from a cephalad movement of the diaphragm due to increased IAP. 20 Finally, the concept of abdominal vascular zones analogous to the pulmonary vascular zone conditions described by West should be emphasized. According to this concept, increased IAP increases venous return when the transmural IVC pressure (defined as IVC pressure minus IAP) at the thoracic inlet significantly exceeds the critical closing transmural pressure (zone 3 abdomen).
Management of Peri-operative Complications, An Issue of Surgical Clinics, 1e by Stanley H. Rosenbaum MD MA, Lewis J. Kaplan